The Aetiology of Deep Venous Thrombosis: A Critical, Historical and Epistemological Survey

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Springer Science & Business Media, Dec 31, 2007 - Medical - 318 pages
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What we now call ‘deep venous thrombosis’ (DVT) has been studied in diverse ways during the last 200–300 years. Each of these approaches contributes to a full modern understanding of aetiology. Therefore, much of this book is a historical survey of the field. However, our remit is broader than the title might suggest: the evolution of ideas about DVT is typical in many ways of medical biology as a whole. Thus, although the aetiology of DVT may seem a narrow topic for a monograph – it implicitly excludes arterial thrombosis and marginalises prophylaxis, therapy, and even such clinically significant sequelae as pulmonary embolism – we hope to engage the reader in a much more general inquiry. Our historical investigation reveals a 160-year-old schism between two contrasting philosophies of medical and biological research, a schism that is particularly – but by no means uniquely – relevant to the study of DVT. In principle, these philosophies should be complementary rather than competing. So while we wish to elucidate the aetiology of DVT per se, we are also concerned with a more abstract and wide-ranging issue: the future accommodation or rapprochement between two conceptual and methodological traditions.
 

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Contents

IV
1
VII
3
VIII
4
IX
7
X
8
XI
9
XII
11
XIII
13
XCVII
133
XCVIII
134
XCIX
135
C
137
CII
138
CIII
139
CIV
143
CV
145

XIV
16
XV
17
XVI
19
XVII
20
XIX
23
XX
25
XXI
27
XXII
30
XXIV
32
XXVI
33
XXVII
34
XXVIII
36
XXIX
37
XXX
38
XXXI
39
XXXII
40
XXXIII
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XXXV
42
XXXVI
44
XXXVII
45
XXXVIII
48
XXXIX
49
XL
51
XLI
54
XLIII
57
XLIV
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XLV
59
XLVII
60
XLVIII
63
XLIX
64
LI
65
LII
66
LIII
67
LIV
71
LVII
72
LVIII
73
LIX
74
LX
75
LXI
77
LXII
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LXIII
81
LXV
83
LXVI
84
LXVII
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LXIX
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LXX
91
LXXI
93
LXXII
95
LXXIII
96
LXXIV
99
LXXV
103
LXXVI
104
LXXVII
106
LXXVIII
107
LXXIX
109
LXXX
110
LXXXI
111
LXXXII
112
LXXXIII
113
LXXXIV
114
LXXXV
115
LXXXVI
116
LXXXVIII
117
LXXXIX
118
XC
119
XCI
120
XCII
122
XCIII
125
XCIV
127
XCV
129
XCVI
132
CVII
147
CIX
148
CX
149
CXI
151
CXII
153
CXIII
154
CXIV
162
CXV
164
CXVI
165
CXVII
166
CXVIII
168
CXIX
169
CXXII
170
CXXIII
171
CXXIV
172
CXXV
173
CXXVI
174
CXXVII
177
CXXVIII
178
CXXIX
183
CXXXI
185
CXXXII
187
CXXXIII
188
CXXXV
189
CXXXVI
190
CXXXVII
191
CXXXVIII
192
CXL
194
CXLII
195
CXLV
197
CXLVII
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CXLVIII
200
CXLIX
202
CL
204
CLII
206
CLIII
207
CLIV
208
CLV
209
CLVI
210
CLVII
211
CLVIII
212
CLIX
216
CLX
218
CLXII
221
CLXIV
222
CLXV
224
CLXVI
226
CLXVIII
227
CLXIX
228
CLXXI
229
CLXXIII
230
CLXXIV
233
CLXXV
234
CLXXVI
235
CLXXVIII
236
CLXXIX
237
CLXXX
238
CLXXXI
239
CLXXXIII
240
CLXXXIV
241
CLXXXV
243
CLXXXVII
245
CLXXXIX
247
CXCI
248
CXCII
249
CXCIII
251
CXCIV
255
CXCV
295
CXCVI
305
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Page 267 - Adenosine diphosphate in red cells as a factor in the adhesiveness of human blood platelets.
Page 256 - Anderson FA Jr, Wheeler HB, Goldberg RJ, et al. A population-based perspective of the hospital incidence and case-fatality rates of deep vein thrombosis and pulmonary embolism. The Worcester DVT Study.
Page 262 - Dahlback B. Carlsson M. Svensson PJ. Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C.
Page 275 - Coexpression of GMP-140 and PAF by endothelium stimulated by histamine or thrombin: A juxtacrine system for adhesion and activation of neutrophils.

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